O risco do tabagismo passivo no desenvolvimento de pneumopatias
##plugins.themes.bootstrap3.article.sidebar##
##plugins.themes.bootstrap3.article.main##
Resumo
Objetivo: Analisar o desenvolvimento de síndromes respiratórias correlacionadas com o tabagismo passivo. Métodos: Trata-se de uma revisão integrativa. Foram utilizadas as bases de dados do portal regional da Biblioteca Regional em Saúde. A busca dos artigos foi feita por meio de descritores, sendo eles “passive smoking”, “risk” e “lung disease”. A revisão foi realizada seguindo as seguintes etapas: estabelecimento do tema, definição dos parâmetros de elegibilidade, definição dos critérios de inclusão e exclusão, verificação das publicações nas bases de dados, análise das informações encontradas, avaliação dos estudos encontrados e exposição dos resultados. Resultados: Foi observado que os danos causados pelo tabagismo passivo podem gerar uma série de consequências negativas para a saúde em geral, como risco para o desenvolvimento de doenças cardíacas e doenças respiratórias e, principalmente, as neoplasias malignas. Esse compilado de fatores, demostra a necessidade do combate ao tabagismo e consequentemente ao tabagismo passivo. Considerações finais: Diante dos fatos mencionados, foi possível inferir que a inalação passiva da fumaça do cigarro aponta para maior risco de agravar ou desenvolver doenças respiratórias, tais como asma, DPOC e câncer de pulmão.
##plugins.themes.bootstrap3.article.details##
Copyright © | Todos os direitos reservados.
A revista detém os direitos autorais exclusivos de publicação deste artigo nos termos da lei 9610/98.
Reprodução parcial
É livre o uso de partes do texto, figuras e questionário do artigo, sendo obrigatória a citação dos autores e revista.
Reprodução total
É expressamente proibida, devendo ser autorizada pela revista.
Referências
2. BASTIAN LA, et al. Differences in Active and Passive Smoking Exposures and Lung Cancer Incidence Between Veterans and Non-Veterans in the Women’s Health Initiative. The Gerontologist, 2016; 56: S102–S111.
3. BLAAKMAN S, et al. Secondhand Smoke Exposure Reduction after NICU Discharge: Results of a Randomized Trial. Academic Pediatrics, 2015; 15(6): 605–612.
4. BOOALAYAN H, et al. Exposure to environmental tobacco smoke and prevalence of asthma among adolescents in a middle eastern country. BMC Public Health, 2020; 20: 1210.
5. BORRELLI B, et al. Motivating parents of kids with asthma to quit smoking: The effect of the teachable moment and increasing intervention intensity using a longitudinal randomized trial design. Addiction, 2016; 111(9): 1646–1655.
6. BUTLER KM, et al. Perceived Synergistic Risk for Lung Cancer After Environmental Report-Back Study on Home Exposure to Tobacco Smoke and Radon. American Journal of Health Promotion, 2019; 33(4): 597–600.
7. BUTZ AM, et al. Children with poorly controlled asthma: Randomized controlled trial of a home-based environmental control intervention. Pediatric Pulmonology, 2019; 54(3): 245–256.
8. BUTZ AM, et al. Factors Associated with Secondhand Smoke Exposure (SHS) in High-Risk Children with Asthma. J Asthma, 2020; 1–12.
9. COURAUD S, et al. BioCAST/IFCT-1002: Epidemiological and molecular features of lung cancer in never-smokers. European Respiratory Journal, 2015; 45(5): 1403–1414.
10. ELSHAZLY FA, et al. Effects of second-hand smoking on lung functions in athlete and non-athlete school-aged children – observational study. Afri Health Sci, 2020; 20(1): 368-75.
11. GRARUP PA, et al. Passive smoking is associated with poor asthma control during pregnancy: A prospective study of 500 pregnancies. PLoS ONE, 2014; 9(11).
12. GROEN E, et al. Evaluation of systematic assessment of asthma-like symptoms and tobacco smoke exposure in early childhood by well-child professionals: a randomised trial. PLoS One, 2014.
13. HANSEN MS, et al. The fraction of lung cancer attributable to smoking in the Norwegian Women and Cancer (NOWAC) Study. British Journal of Cancer, 2021; 124: 658–662.
14. HASSANZAD M, et al. Cotinine Level Is Associated with Asthma Severity in Passive Smoker Children. Iran J Allergy Asthma Immunol, 2015; 14(1): 67-73.
15. HUNTINGTON ML, et al. Radon, Secondhand Smoke, and Children in the Home: Creating a Teachable Moment for Lung Cancer Prevention. Public Health Nursing, 2016; 33(6): 529–538.
16. HUNTINGTON ML, et al. Use of theory-driven report back to promote lung cancer risk reduction. International Journal of Environmental Research and Public Health, 2021; 18(20).
17. HUTCHINSON SG, Motivational interviewing, and urine cotinine feedback to stop passive smoke exposure in children predisposed to asthma: a randomised controlled trial. Scientific reports, 2017; 7: 15473.
18. MOHAMMAD Y, et al. Impact of active and passive smoking as risk factors for asthma and COPD in women presenting to primary care in Syria: first report by the WHO-GARD survey group. International Journal of COPD, 2013; 8: 473-482.
19. PEREZ AV, et al. Characterization of severe asthma in the pediatric population. Allergol Immunopathol (Madr), 2021; 49(2): 60–65.
20. PIAZZA KM, et al.Inhaled medication usage in post-menopausal women and lifetime tobacco smoke exposure: The Women’s Health Initiative Observational Study. Maturitas, 2016; 90: 42–48.
21. UZELOTO JS, et al. Mucociliary Clearance of Different Respiratory Conditions: A Clinical Study. International Archives of Otorhinolaryngology, 2021; 25(1): e35–e40.
22. VIÑOLAS N, et al. Lung Cancer in Never-Smoking Women: A Sub-Analysis of the Spanish Female-Specific Database WORLD07. Cancer investigation, 2017; 35: 1-8.
23. WAGENER TL, et al. Changes in risk perception following a smoking cessation intervention: the role of acculturation in a sample of Latino caregivers. Journal of Behavioral Medicine, 2014; 37(5): 1000–1008.
24. WANG A, et al. Active and passive smoking in relation to lung cancer incidence in the Women’s Health Initiative Observational Study prospective cohort. Annals of Oncology, 2015; 26(1): 221–230.
25. ZUBAIR T, et al. Role of passive smoking in non-smoking related chronic obstructive pulmonar disease. JPak Med Assoc, 2018; 68: 1310-1315.